Dogs are kept at home, or when the owner takes the dog for a walk, the dog may eat indiscriminately if he is not careful, and food poisoning may occur if he is not careful. It can be big or small, a little better, as long as you take the dog to the pet hospital for treatment, it is almost the same, but there are also many dogs who die because of eating by mistake! So when dogs eat by mistake , we have to learn to be calm and deal with it calmly!

Food Poisoning

Dogs ingested spoiled After fish, meat, yogurt and other foods, these spoiled foods contain higher amounts of Proteus, Staphylococcus toxins, Salmonella enterotoxins and Clostridium botulinum toxins. Deteriorated fish is contaminated with Proteus, which causes protein breakdown and produces histamine. The incubation period of histamine poisoning is not more than 2 hours, the dog suddenly vomits, diarrhea, dyspnea, nasal discharge, mydriasis, ataxia, the dog may coma, hindquarters paralysis, weakness, hematuria, black stool. In the treatment of spoiled fish poisoning, intravenous or subcutaneous injection of glucose and vitamin C, oral administration of diphenhydramine, intramuscular or subcutaneous injection of penicillin

Staphylococcal toxin poisoning can cause symptoms of acute gastroenteritis, vomiting, abdominal pain, Diarrhea. Difficulty breathing, convulsions, and convulsions occur in severe cases. Emetic induction, fluid replacement and symptomatic treatment were used during treatment. Gastric lavage and enema can be performed if necessary. Clostridium botulinum can cause motor paralysis, coma, and even death in dogs. Symptoms in dogs of Clostridium botulinum poisoning are related to ingestion. In the initial stage, the neck and shoulder muscles were paralyzed, and then quadriplegia gradually appeared, with unresponsiveness, dilated pupils, difficulty swallowing, saliva flow, and drooping ears. The eyes are more common with conjunctivitis and ulcerative keratitis. He eventually died of respiratory paralysis. Clostridium botulinum poisoning has a short course of disease and a high mortality rate. Antitoxin is injected immediately after onset, intravenously or intramuscularly. Symptomatic treatment can be gastric lavage with 0.01% potassium permanganate solution, laxative or enema, intravenous infusion, and intramuscular injection of penicillin. The best way to prevent this disease is that the dog's food should be cooked and not stored for a long time.

Poisoning by rodenticide

1. Poisoning by antirodent drugs. This is a powerful rodenticide. It is a white, odorless, crystalline powder, which increases the permeability of pulmonary capillaries and causes a large amount of plasma to enter the lung tissue, resulting in pulmonary edema. After a few minutes to a few hours after the dog ingests, the dog will vomit, foam at the mouth, then diarrhea, cough, difficulty breathing, depression, visible mucous membrane cyanosis, and foamy blood-colored mucus from the nostrils. Generally, coma and lethargy occur 10-12 hours after ingestion, and a few die within 2-4 hours after ingestion. There is no specific antidote for this poisoning, and the methods of emesis, gastric lavage, catharsis and diuresis can be used.

2. Zinc phosphide poisoning. This is a commonly used rodenticide and is a grey powder. A few days after ingestion, it mixes with water and stomach acid in the stomach, releasing phosphine gas, causing severe gastroenteritis. The sick dog has abdominal pain, no food, continuous vomiting, coma and lethargy, and rapid and deep breathing. Choking, diarrhea, blood in stool. Treatment can be administered with 0.2%-0.5% CuSO4. 10-30 ml of solution to induce vomiting and expel toxins in the stomach. Gastric lavage can be used 0.02% KMnO4 solution, and then with 15 grams of Na2SO4 catharsis. Intravenous injection of hypertonic glucose solution is beneficial for liver protection.

3. Organofluoride rodenticide poisoning. This is a highly poisonous drug. After 2-3 days of eating, the sick dog becomes restless, vomits, has gastrointestinal hyperactivity, runs around, barks, and has paroxysmal convulsions all over the body, lasting about 1 minute, and finally death. For the treatment of intramuscular injection of fluring, 0.1-0.2 grams per kilogram of body weight, the first dose is 1/2 of the daily dose, and the remaining 1/2 dose is divided into 4 parts, which are injected every 2 hours. In combination with emesis and gastric lavage. Feeding raw egg whites to sick dogs will help protect the mucous membranes of the digestive tract. Intravenous calcium gluconate 5-10 ml is beneficial.

Organophosphorus pesticide poisoning

Organophosphorus is widely used in agriculture as pesticides, such as trichlorfon, dimethoate, dichlorvos and so on. Accidental ingestion can cause a lot of salivation, tearing, diarrhea, abdominal pain, urinary incontinence, dyspnea, cough, cyanosis of the conjunctiva, muscle twitching, and then paralysis, miosis, and coma. Most of them died due to breathing disorders. During treatment, firstly, atropine sulfate was slowly injected intravenously, 0.05 mg per kilogram of body weight, and 0.15 mg of atropine sulfate per kilogram of body weight was injected subcutaneously or intramuscularly after an interval of 6 hours. Phosphatidyl can enhance the function of atropine. Medications to relieve muscle spasms that help ease symptoms. Individual dogs are allergic to pyridoxine and chlorophosphine, and attention should be paid to them.

Poisoning by chlorinated hydrocarbons

Such pesticides include DDT, hexahexahexanol, etc. Cause the dog to be extremely excited, manic, restless or highly depressed, the head and neck muscles first tremble, then spread to the whole body, muscle spasms and contractions, and then depression; continuous birth, no or little food, diarrhea. Treatment can be performed by cleansing and gastric lavage, followed by catharsis with salt laxatives. Symptomatic treatment of hyperexcitability in dogs is given with sedatives. Because the dog is dehydrated and does not eat, intravenous fluids should be given.